1	Adenosine		_____	ventricle not pumping effectively, insufficient cardiac output
2	Atropine		_____	synchronized cardioversion, IV amiodarone, tx underlying cause
3	Dobutamine		_____	death or gradual recovery
4	Dopamine		_____	inotropic, inc CO, BP and renal perfusion, for brady cardia, hypotension and poor cardiac output
5	Epinephrine		_____	antiarrhythmic, administer rapidly, for SVT
6	Glucose		_____	acetylcystenine (Mucomyst)
7	Lidocaine		_____	used for SVT, V-tach with a pulse, synchronized on R waves
8	Narcan		_____	hepatic stage, pain in upper right quadrant
9	CPR		_____	one person 30:2, two person 15:2, fingers and thumb at the nipple line
10	CPR infant		_____	estimates wgt of child based on length
11	CPR child		_____	anticholinergic, inc CO, dries secretions, for sinus bradycardia, asystole, PEA, don't mix with sodium bicarbonate
12	Defibrillation		_____	catecholamine, inc myocardial contractility and HR, mgt of shock
13	Cardioversion		_____	no palpable pulse or signs of perfusion, Asystole, PEA
14	Automated External Defib		_____	fever, pain, fear; physiologic causes such as hypoxia and hypovolemia, focus on underlying cause
15	Broselow tape		_____	vagal maneuvers such as ice to face of blowing through a straw, adenosine
16	Shock		_____	HR <60 and signs of altered perfusion
17	Hypovolemic shock		_____	antagonizes action of narcotic agents, watch for respirator despression
18	Septic shock		_____	caused by impaired cardiac output or impaired SVR
19	Septic shock: warm shock		_____	result in loss of SVR; hypovolemia occurs with neurogenic injury-related shock and anaphylaxis
20	Septic shock: cold shock		_____	systemic inflammatory response
21	Cardiogenic shock		_____	adenosine or synchronized cardioversion
22	Distributive shock		_____	inc CO and low SVR
23	Altered microcirculatory status		_____	compensatory mechanisms activated in response to decreased blood flow
24	Bradyarrhythmias: Sinus bradycardia		_____	determine if SVT or VT
25	Bradyarrhythmias: Bradycardia related to heart block		_____	indicated for childern over 1 who have experienced sudden witnessed collapse and have no pulse
26	Bradyarrhythmias: Serious, life threatening		_____	regular rhythm and extremely rapid rate, problem with conduction system, genetic problem
27	Bradyarrhythmias management		_____	used to depolarize myocardial cells to terminate abnormal life threatening rhythm, used in conjunction with oxygen, CPR and medications
28	Tachyarrhthmias: Sinus tachycardia		_____	systemic perfusion decreses due to inadquate intravascular volume, occurs in association with fluid losses
29	Tachyarrhthmias: SVT		_____	one person 30:2, two person 15:2, heel of hand or two hands pressing on sternum at the nipple line
30	Tachyarrhthmias: VT		_____	elevated HR and wide QRS; abnormal rapid firing of ventricles
31	Tachyarrhthmias management		_____	oxygen, ventilate, EPINEPHRINE, ATROPINE, antidotes to toxins
32	Tachyarrhthmias management Compensated SVT		_____	HR < 60 bpm, two person no pauses for ventilation
33	Tachyarrhthmias mnagement Uncompensated SVT		_____	dec CO and high SVR
34	Tachyarrhthmias mnagement VT		_____	antidysrhythmic, dec automaticity of conduction tissues of heart, for ventricular arrhythmias, contraindicated in complete heart block
35	Collapsed rhythms (pulse-less rhythm)		_____	inc blood glucose level, for hypoglycemia
36	V Fib		_____	adrenergic, inc HR and SVR, for bradycardia, anaphylaxis
37	Acetaminophen Overdose: 3-7 days after or longer		_____	brief dips can be normal such as when child has vagal response; recovers with or without stimulation and no signs of altered perfusion
38	Acetaminophen Overdose: Final stage		_____	ineffective pumping of the hear causing decrease in stroke volume
39	Acetaminophen Overdose intervention		_____	exhibit poor feeding, tachypnea in young children, fatigue, dizziness, and syncope in older children